Cardiac inflammation, myocardial infarction, heart failure, cardiocrine signaling, development of nanoparticle-based treatment approaches.

2022 Arthur Weber Price of the German Cardiac Society (DGK)

2021 European Coordinator Leducq Network “The Inflammatory-Fibrosis Axis in Ischemic Heart Failure”

2021 Albert Fraenkel Price of the DGK

2020 Wilhelm P. Winterstein-Award of the German Heart Research Foundation

2018 Heisenberg Professorship “Immunocardiology” (W3 Full Professorship), University of Heidelberg

2018 Head of Interventional Valve Replacement, Univ. Hospital Heidelberg

2016 Science Award Walter Siegenthaler Society

2015 Member and Speaker of the Junior Research Group of the Federal Ministry of Education and Research (BMBF, DeCaRe Consortium)

2013 Member/ Principal Investigator of the German Centre for Cardiovascular Research (DZHK, Partner Site HD/MA)

1. Sicklinger F, Meyer IS, Li X, Radtke D, Dicks S, Zhang Y, Kuhn TC, Patel J, Boerries M, Schramm G, Frey N, Katus HA, Voehringer D, Leuschner F. (2021) Basophils balance healing after myocardial infarction via IL-4/IL-13. J Clin Invest. 131(13):e136778.

2. Sicklinger F, Zhang Y, Lavine KJ, Simon N, Bucher V, Jugold M, Katus HA, Leuschner F. A minimal-invasive approach for a standardized induction of myocardial infarction in mice. (2020) Circ Res. 127:1214-16.

3. Kuhn T, Knobel J, Burkert-Rettenmaier S, Li X, Meyer IS, Jungmann A, Sicklinger F, Backs F, Lasitschka F, Müller OJ, Katus HA, Krijgsveld J, Leuschner F. (2020) Secretome analysis of cardiomyocytes identifies PCSK6 as a novel player in cardiac remodeling after myocardial infarction. Circulation. 141(20):1628-1644.

4. Leuschner F, Nahrendorf M. (2019) Novel functions of macrophages in the heart: insights into electrical conduction, stress, and diastolic dysfunction. European Heart Journal. 41(9):989-994.

5. Meyer IS, Jungmann A, Dieterich C, Zhang M, Lasitschka F, Werkmeister S, Haas J, Müller OJ, Boutros M, Nahrendorf M, Katus HA, Hardt SE, Leuschner F. (2017) The cardiac microenvironment uses non-canonical WNT signaling to activate monocytes after myocardial infarction. EMBO Molecular Medicine. 9(9):1279-1293.